|  Author | Topic: mFadA nuclear struct/spiro/filaments/spores (Read 342 times) |
skyship
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« Thread Started on Aug 25, 2011, 7:12pm » | ![[Quote]](http://images.proboards.com/buttons/quote.gif "[Quote]") |
Could the nucleus of the fusobacterium nucleatum been used to form the filament?
http://www.jbc.org/content/284/6/3865/F8.large.jpg
"Some packing interactions with neighboring molecules in the variant FadA crystal structures are different from wild type and from each other (Fig. 6). However, the head-to-tail interaction between neighboring molecules observed in the wild-type crystal structure (Fig. 7) is conserved in all the variant structures, suggesting that the head-to-tail interaction represents a physiologically relevant oligomerization motif.
Filament Formation of FadA and Its Variants—Electron microscopy studies revealed formation of long filaments by purified wild-type FadA (Fig. 8). The filaments varied in width, ranging from 30 to 170 Å, as well as in length. L14A and L76A exhibited no filaments (Fig. 8). "
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...."The crystal structure of mFadA reveals a quintessential role for leucine residues, which mediate both intramolecular and intermolecular interactions. First, they comprise the main “molecular glue” to associate the two helical arms of the monomer; second, they form the head-to-tail association of monomers to form the antenna-like polymers."...........
http://www.jbc.org/content/284/6/3865.full
So, this tells me the molecular glue connects to the arms of monomer,
and they form ....."antenna-like polymers............
Monomer makes oligomer........
Skyship
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kritters
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« Reply #1 on Aug 29, 2011, 1:47pm » | |
Aug 25, 2011, 7:12pm, skyship wrote:Could the nucleus of the fusarium have been used to form the filament?
http://www.jbc.org/content/284/6/3865/F8.large.jpg
"Some packing interactions with neighboring molecules in the variant FadA crystal structures are different from wild type and from each other (Fig. 6). However, the head-to-tail interaction between neighboring molecules observed in the wild-type crystal structure (Fig. 7) is conserved in all the variant structures, suggesting that the head-to-tail interaction represents a physiologically relevant oligomerization motif.
Filament Formation of FadA and Its Variants—Electron microscopy studies revealed formation of long filaments by purified wild-type FadA (Fig. 8). The filaments varied in width, ranging from 30 to 170 Å, as well as in length. L14A and L76A exhibited no filaments (Fig. 8). "
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...."The crystal structure of mFadA reveals a quintessential role for leucine residues, which mediate both intramolecular and intermolecular interactions. First, they comprise the main “molecular glue” to associate the two helical arms of the monomer; second, they form the head-to-tail association of monomers to form the antenna-like polymers."...........
http://www.jbc.org/content/284/6/3865.full
So, this tells me the molecular glue connects to the arms of monomer,
and they form ....."antenna-like polymers............
Monomer makes oligomer........
Skyship
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Hi, Sky~
Okay, so this is really funky. This morning, I read report by a website I get news from all about how Glyphosate (Roundup) is causing fusarium proliferation in crops and is killing not only bananas, but other crops as well (as we all know here).
I apologize for not having been on 'board' for a bit, but have been working on my daughter's bridal shower I was to have here Sunday, until I had to prepare for Hurricane Irene. Well, fortunately, we can sing, "good night Irene" and now I'm back putting back furniture on the decks and unboarding the windows.
Anyway, I figured there would be something about fusarium, glyphosate, roundup here on the forum, so did a search and your very recent post just came up.
Here's what I wrote to my family and friends in an email:
PLEASE READ THIS:
http://healthfreedoms.org/2011/08/29/mon....es-in-the-mine/
"...New information from Scientific American indicates that Monsanto’s glyphosate (Roundup) literally encourages the growth of Fusarium fungus at the roots of plants..."
"...Fusarium is a large genus of filamentous fungi widely distributed in soil and in association with plants. Most species are harmless saprobes, and are relatively abundant members of the soil microbial community. Some species producemycotoxins in cereal crops that can affect human and animal health if they enter the food chain. The main toxins produced by these Fusarium species are fumonisins and trichothecenes..."
(one reason way overlooked, you should not let children walk or play in soil)
http://www.lawyersandsettlements.com/case/eye_contacts_infection.html
Brings to mind my frequent eye infections. I use that lens solution. I no longer use my contacs and my vision is not bad now. Fusarium is most likely causing these infections.
http://www.mold-help.org/content/view/417
Fusarium is listed as one capable of causing mycetomas, and it has repeatedly been isolated from human keratitis and corneal ulcers. Most cases concern keratitis.
I had an eye infection that was so bad, I know I have corneal ulcers. I may have written about my latest last month. so they've found out this cleaning, storing solution causes these problems. I haven't put contacs in and my eyesight is strangely improved. That infection was so painful, it was like I had class in my eye for 2 days. I know it was fungal related.
So, what do you say....fusarium fungus?
Kritts
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skyship
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« Reply #2 on Aug 29, 2011, 4:12pm » | |
Kritters,
There is more than just the keratitis and eye...............
"A novel adhesin protein, FadA, was recently identified from the opportunistic human pathogen Fusobacterium nucleatum (2). F. nucleatum is one of the most abundant Gram-negative anaerobes colonizing the subgingival plaque, present in both healthy and diseased periodontal sites and associated with various forms of periodontal disease (3). It has been suggested that F. nucleatum migrates from its primary site of colonization in the oral cavity to other parts of the body via hematogenous transmission.
Transmission of F. nucleatum into amniotic fluid and placenta of pregnant women causes premature delivery (4). Animal studies have shown that, once in the bloodstream, F. nucleatum specifically colonizes the placenta and activates TLR4-mediated placental inflammatory responses, leading to preterm and term stillbirths and unsustained live births (5, 6). F. nucleatum binds to and invades both epithelial and endothelial cells, causing host inflammatory responses (2, 7), and FadA is required for bacterial attachment and invasion of the host cells (2, 8).
Here it is:
==================
http://www.sciencephoto.com/media/11402/enlarge
skyship
Fusobacterium:
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skyship
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kritters
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« Reply #4 on Aug 29, 2011, 4:50pm » | |
Sky,
What is the association between the fusarium fungus and the fusobacterium bacteria? I tried to see if they are a combo of fungus and bacteria, but the 'fus' is related to the spindle shape, in the later case.
One is a fungus and the other a bacteria. 
I bet it's not just in sinus and periodental areas (as mine are. remember I just had two implants on teeth which are in maxillary sinus area and are symmetrical. It affects my eyes, nose, teeth and I'm SURE ears.
Thanks for response!
Kritts
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skyship
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« Reply #5 on Aug 29, 2011, 8:55pm » | |
It also effects pregnancies. that particular strain.
It links to chlamydias. It is a conjugation of a bacteria, virus most likely and a fungi.
that is why localizes in nose area, mouth.
In this case since it is a nucleatum, it could be in the nucleus of the fusarium fungi cell ?
FadA is the bacterium, while fusiforium fungi is different, it is just the fungi but still can conjugate
with other fungi and yeast, espec candida.
http://www.broadinstitute.org/annotation..../MultiHome.html
Sky ship
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kritters
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« Reply #6 on Aug 29, 2011, 9:40pm » | |
Sky, you're amazing.
So, I reckon it's possible Dr Moreau and co. has figured out how to interbreed these things into something they thought no one would figure out.
Sorry, Doc, seems our members of this forum are on it.
Tee hee
xoKritts
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kritters
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« Reply #7 on Aug 29, 2011, 9:42pm » | |
seems to me also that if we know what kills fungi and also strengthens our immune systems, we might just knock this sucker out.
Kritts
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skyship
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skyship
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« Reply #9 on May 15, 2012, 11:52pm » | |
Fusobacterium nucleatum Transports Noninvasive Streptococcus cristatus into Human Epithelial Cells
Examination of KB monolayers incubated with both F. nucleatum and S. cristatus revealed the direct attachment of streptococci to F. nucleatum bacteria that were in the process of internalization
http://iai.asm.org/content/74/1/654/F3.full
Spheres, rods, filaments, spheres forming filaments etc.....
On the side this is mentioned leptospirillium
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Leptospiral Flagellar Sheaths and FlaA Proteins
It has been accepted that the sheath surrounding the periplasmic flagella of spirochetes is composed of FlaA proteins. Indeed, that has been true for spirochete genera studied to date. Lambert et al. (p. 2019–2025) now show that Leptospira interrogans is different. Inactivation of the two leptospiral flaA genes has no effect on sheath morphology. However, while the flagellum sheath is unchanged, the mutant has altered flagellum helicity, lacks the usual hook-shaped ends, loses translational motility, and is attenuated for virulence. These results demonstrate that FlaA proteins contribute to flagellum function and confirm that motility is essential for virulence.
http://iai.asm.org/content/80/6/1947
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blebbing of the fusobacterial membrane was observed for the majority of the bacteria (A, B, E, and F). There was evidence of intercellular migration by F. nucleatum (C). S. cristatus was rarely observed to adhere directly to epithelial cells; attachment occurred via polar tufts of fibrils
http://iai.asm.org/content/74/1/654/F4.expansion.html
Here is biofilm formation in mouth: 5 species plus t. denticola spirochete, possibly 2 others.
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5 important physiological-, cariogenic-, and periodontitis-associated microorganisms (Streptococcus sanguinis, Streptococcus mutans, Fusobacterium nucleatum, Aggregatibacter actinomycetemcomitans, and Porphyromonas gingivalis)
http://www.ncbi.nlm.nih.gov/pubmed/22194794
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skyship
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« Reply #10 on May 16, 2012, 12:21am » | |
The thing about fusobacterium is it is intercellular......
http://www.ncbi.nlm.nih.gov/pmc/articles....22034511399096/
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Is it collagen or keratins that are effected? This is in the saliva, which is in other areas.
It begins in the mouth, anyway you look at it. the gut, the lungs, the soft tissue, the blood,
fibronectin is damaged
epithelium is damaged
and appears the collagen is.
these are all over the body, involves the cells.........
mouth: highly organized spirochetes:
These clusters of spirochetes are a strange phenomena. We do not know what this behavior is about.These spirochetes are microscopically indistinguishable from syphilis or lyme disease spirochetes. Oral spirochetes have been found in the brains of alzheimer's patients. We believe that oral spirochetes are the primary injurious agent in two other chronic diseases that plague man, heart disease and diabetes. These things breed by the trillions in the gingival sulcus and invade into the body by millions moving via the de-epitheliazed gingival sulcus into the blood stream then into cells found along the blood stream.
Primarily the endothelial cells lining blood vessels, and the Islets of langerhans cells in the pancreas.. We have not seen anyone with heart disease or diabetes who are not infected with oral spirochetes. Recent papers have proven the alzeheimers plaques are created by these spirochetes which breed in the crevice between the tooth and the gum and under plaque bacteria. The use of tooth cleaning agents will not remove these spirochetes.
The only effective methods we have found is Dakins solution. Vigorous rinses for at least two minutes with Dakins or Dakins in a WaterPic®. The use of the Dakins which is a 20:1 dilution of clorox bleach is by far the most effective technique for killing spirochetes in between the teeth as well as the more accessible areas. Tooth pastes are good for cleaning teeth! But this is a different problem entirely. Spirochetes form spores <b>...</b>
http://wn.com/dental_plaque
Anyone used Dakins solution?
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skyship
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« Reply #11 on May 16, 2012, 12:39am » | |
Proof that the various spirochetes are causing much damage, one of them t. denticola spirochete,
So, if the fusobacterium causes the plaque, where is it from? How does it get into teeth with the
spirochetes?
Relationship of spirochetes to fusobacterium: I believe the interspecies mix of these two is one of the problems: fusobacterium blebbs on the spirochete. forms a sphere on the spirochete coat.
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Spirochete Spore like Forms Found inside the Cells of the “GUMS” Around Teeth
"When studying the old literature when microbiology was at its “hay-day” (pre-1920’s), it seems that these astute microbiology scientists understood the fact that spirochetes, unlike most other bacteria, have many forms better then we recognize today. Krutchkoff[2], when presenting the data and literature review from the book, The Silent Saboteur, he coauthored to his fellow professors, Deans of three dental schools, astute colleagues and longtime academic friends found a militant rejection of the basic principle of pleomorphism, “spore-like” forms and cell-wall deficient forms of spirochetes even though, as will be presented here, there are references to these facts that go back almost one hundred years proving that these forms exist. It’s hard to understand why dentistry doesn’t want to expose these facts to the public."
http://lifeguardyourhealth.com/spirochete-spore-like-forms/
I think we are getting closer.
skyship
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skyship
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« Reply #12 on May 16, 2012, 12:45am » | |
So, this covers MS, Alzheimers (plaque formed, same in Alz), ALS, Lymes, Morgellons is what then?The oligomerization of this: Uses the coat protein from the spirochete, with the fusobacterium, a aggregation, with the p. gingivalis forming form the plaque forming fusobacterium, T denticola is one of the spirochetes.Now, does this now sound similar to what the Morgie children were talking about? The spirochete in the hoof of the cow, similar to Morgellons. Why the link?
HOOF and MOUTH disease, it has been around a long time............
http://lifeguardyourhealth.com/wp-content/uploads/2011/05/EM-spore1.jpg
http://lifeguardyourhealth.com/wp-conten....n-to-spore1.jpg
http://lifeguardyourhealth.com/wp-conten....kld-spores1.jpg
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skyship
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« Reply #13 on May 16, 2012, 12:56am » | |
Spirochete warfare:
"An article by Marjorie Tietjen called "Discreet Methods of Biological Warfare" offers some interesting points and some food for thought.
In her article she mentions Lyme disease and whilst a supposedly "old" disease, has a somewhat curious genetic structure that looks like it "was captured in mid-shuttle," like it is "still undergoing construction."
Let´s explain this disease more (information obtained from MedicineNet.com). Lyme disease is a bacterial illness caused by a bacterium called a "spirochete" In the United States, the actual name of the bacterium is Borrelia burgdorferi. In Europe, another bacterium, Borrelia afzelii, also causes Lyme disease. Certain ticks found on deer harbor the bacterium in their stomachs. Lyme disease is spread by these ticks when they bite the skin, which permits the bacterium to infect the body. Lyme disease is not contagious from an affected person to someone else, and the disease can cause abnormalities in the skin, joints, heart, and nervous system. (According to Alan MacDonald, M.D Lyme disease can also be passed on by 'vertical transmission' from mother to fetus)
Interestingly, the disease only became apparent in 1975 when mothers of a group of children who lived near each other in Lyme, Connecticut, made researchers aware that their children all were diagnosed with rheumatoid arthritis. This unusual grouping of illness that appeared "rheumatoid" eventually led researchers to the identification of the bacterial cause of the children's condition, what was then called "Lyme disease" in 1982.
Lyme disease affects different areas of the body in varying degrees as it progresses. The site where the tick bites the body is where the bacteria enter through the skin. Initially, the disease affects the skin, causing an expanding reddish rash often associated with "flu-like" symptoms. Later, it can produce abnormalities in the joints, heart, and nervous system.
Lyme disease is medically described in three phases as: (1) early localized disease with skin inflammation; (2) early disseminated disease with heart and nervous system involvement, including palsies and meningitis; and (3) late disease featuring motor and sensory nerve damage and brain inflammation and arthritis.
One of the interesting areas of Lyme disease is the "three phases" and the following article by Alan MacDonald, M.D. is highly recommended. Unfortunately, there is too much information to include all of it here, so I shall just add a few extracts.
Dr. MacDonald became interested in Alzheimer's disease and Lyme when attending a Lyme disease conference in Vienna, Austria. "The speakers were describing Lyme disease as having three stages: primary, secondary and tertiary. Since those terms are also used to describe syphilis, it occurred to him that if Lyme disease had a tertiary, late-stage form, perhaps the late manifestation could be dementia. Like syphilis, which also had late stage dementia, Lyme disease may reoccur thirty or forty years after infection. He hypothesized that if there is a dementia occurring in late-stage Lyme, it might be an Alzheimer's-like illness, a late manifestation of bacterial infection in the brain."
Dr. MacDonald explains how "spirochetes have more diverse forms than just the corkscrew, or spiral form." And this explains why many doctors (and the commercially available tests) are unable to discover the disease. As Dr. MacDonald says "If you're looking for evidence of an infection in tissue you want to see the bug. And if you're looking for the bug and you only look for spiral forms, you may miss the other forms, forms that are granular or rounded cyst forms or L-forms."
In another article by Elena Cook "Spirochete Warfare" she says "Borrelia, the microbes which cause Lyme disease, are a sub-type of the wider biological classification of spirochetes. Now it has become apparent that the spirochetes were weaponized over 75 years ago."
Again, this is quite a long article and we would suggest using the link to read all the information, but here is a small extract from the article:
"We now know, for example, that the US allowed leading Nazi bioweaponeer Erich Traub to play a major role in setting up research at their biowarfare lab on Plum Island, a stone's throw away from Lyme, Connecticut, where the first recorded outbreak of Lyme disease in America occurred in the 1970's. Traub's germ warfare knowledge was considered so important that, his Nazi past notwithstanding, he was invited to take charge of scientific research on the Island in the 1950's.""
It may be of interest to the public that the research complex on Plum Island, about 1.5 miles off the eastern shore of New York's Long Island, is almost certain to be relocated to a new site in Kansas (Government recommends Kansas for biodefense lab). "The Bush administration acknowledged this year that accidents have happened" on Plum Island and one must wonder at the logic of bringing such a complex to the mainland.
Although "officially" the new lab will study foot-and-mouth disease, African swine fever, Japanese encephalitis, Rift Valley fever and the Hendra and Nipah viruses, there is no way of telling what else may be included.
Biological Warfare can be used in many ways, it can be developed to kill selected races, disable a country by "infecting" large numbers of the population, birth control and perhaps even making the public more susceptible to authority?
It would be easy to dismiss these possibilities as science fiction, but with the destructive force of nuclear weapons already abundant on our planet, we know our leaders are capable of producing (and using) such methods, even though they put our very existence at risk. Sadly, the country that lectures others the most about nuclear and biological weapons (and leads in both of these fields), is the United States, a country that has the dubious "honor" of being the first (and so far only) nation to ever use a nuclear bomb on an enemy.
With this thought in mind, we should remember that any country with the capability of using biological weapons is a danger and no one is immune from the temptation of putting such destructive weapons to use. It is up to us (the public) to guard against this madness, because we are the ones that will pay the ultimate price.
http://www.americanchronicle.com/articles/view/84139
So, I do believe they found an adequete disease causing group of pathogens to fulfill the purpose
of this particular plague.
At some point we will survive, we will become immune to them, but if we get anywhere near
those who do not have it, we shall kiss them, hug them and thank them for being so concerned about us. Just make sure they are "sloppy saliva" kisses as well.
Skyship
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skyship
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« Reply #14 on May 16, 2012, 1:11am » | |
The dentists have seen them. all the forms. and the movers.
So, how does the oligomerization take place with the fusobacterium and the spirochete coat? the same way it does in Alzheimers, it begins in the teeth. gut or blood. the fibronectin,
the epethelial cells and collagen. One thing about the teeth is that the peridontal ligand is eaten away. So the ligand around the tooth roots is eaten away.. No protection from the fusobacterium causing the plaque.
What causes plaque in the first place?
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The morphology of Borrelia is similar to that of other medically important spirochetes, including the syphilis-causing Treponema pallidum and the leptospirosis-causing Leptospira interrogans (Holt, 1978). The cytoplasm of spirochetes is surrounded by a typical bacterial plasma membrane, and a second outer lipid bilayer membrane or membrane sheath with an external amorphous slime layer (Hovind-Hougen, 1976; Barbour and Hayes, 1986). The periplasmic space between both membranes contains the peptidoglycan layer and flagellar filaments. The flagella, which are important for both maintaining cell shape and motility (Charon and Goldstein, 2002), originate from basal bodies located on the cytoplasmic cylinder close to both ends forming two bundles that overlap in the centre of the cell (Johnson et al., 1984).
Spirochetes can move forward and backward, and flex the body in stationary phases when changing direction (Charon and Goldstein, 2002). Cytoskeletal elements were found in the form of CfpA in Treponema denticola (Izard et al., 1999). However, little is known about the function of other bacterial filamentous structures, such as the bacterial tubulin homologue FtsZ (filamentous temperature-sensitive protein Z) or the bacterial actin homologue MreB.
http://onlinelibrary.wiley.com/doi/10.1111/j.1365-2958.2009.06613.x/full
mFadA nuclear structure
Cfpa cytoskeletal elements
FtsZ: bacterial filementous structures homologue(a look alike)
MreB: bacterial actin homologue(a look alike)
Protein Z: filamentous temperature-sensitive protein Z)
Skyship
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skyship
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« Reply #15 on May 16, 2012, 4:43pm » | |
If you have spirochetes in mouth:
Please see video of no spirochetes.
http://www.youtube.com/watch?v=xnZ8KylZneM&feature=endscreen&NR=1
MRG is not endorsing this, just putting information out there for you to consider. If you do not understand its use, please contact the doctors who understand how to use this.
==================================
This is the phase contrast slide of a healthy human... 88 years old. No systemic illnesses, no heart disease, no diabetes, no mental deficit. No periodontal disease. This man is immune to spirochetes. The slide of a very damaged caries area, shows a person without spirochetes. We have been monitoring our very old patients for the last 5 years and found that our very old population who are healthy with no chronic illnesses have no spirochetes.
Our in house study is up to over 100 patients who have no chronic illnesses. These folks have not had diabetes, heart disease, or signs of alzheimers. They have no oral spirochetes! Why? We do not know why some persons are not infected with spirochetes but we are seeing that those who are not have no other chronic illnesses as well. These persons are remarkable... The question for the rest of us who are infected with spirochetes is can we alter the outcome of our lives by elimninating the spirochetes we breed in the nursery
called the gum crevice.
If we can eliminate spirochetes from growing in the safe habitat of the gum crevice can we alter the future of other chronic diseases such as heart disease, diabetes, and alzheimers. I believe the answer is yes. The oral spirochetes proliferate only in the gum crevice. They invade and do acts of terrorism over a life time.. If we kill them in the save haven of the gum crevice can we be safe?
Probably only if we maintain a constant vigilance of daily oral disinfection with Dakins a 20;1 dilution of clorox. Unfortunately we do not all have a mouth such as this naturally. But we can create it looking like this with diligence using oral rinses with bactericidal materials. This patient who had tooth decay which is best treated using tooth pastes, and effective brushing and flossing, did not have leaking seals. (bleeding gums)
Here is the recipe for Dakins: please be careful using this though.
http://www.virginia.edu/uvaprint/HSC/pdf/09024.pdf
http://woundconsultant.com/files/DakinsRecipe.pdf
skyship
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1lunker
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« Reply #16 on May 16, 2012, 10:31pm » | |
would not gargling with h2o and a small sip of vinegar, then thoroughly rinsing with h20 twice, do the same thing? Does maintaining a slightly acidic mouth limit spirochetes?
test for me; full mouth of water and small sip vinegar and rinse, at night before bed. What's your breath like in the morning?
next night, brush teeth with just baking soda and rinse. What's your breath in the morning?
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skyship
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« Reply #17 on May 16, 2012, 11:35pm » | |
Lunker,
good test. That would show us something, or tell us how the plaque keeps forming?
make it simple. for sure. Might be all we need.
will give it a try.
Thanks for reminding us again of some of the most simple products. Doesn't have to be as complicated as the Spirochete or fusobacterium link, but wonder what it will do to the polymer
oligomer. Might just eat that too? Does vinegar eat resin?
Since citrus remove the glue spray can remove glue off bottles where labels have been. Could even the shavings of oranges, grapefruit or even lemon assist in breaking down the resin oligomer?
mmm another test to try. Next time I have a filament will put it to the test, see how it reacts. Of course if they are made of some kind of vinyl carbon mix, I don't know. The polymer could be broken down
I bet if it is made of resin?
Skyship
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skyship
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« Reply #18 on May 16, 2012, 11:46pm » | |
Some kind of thiophenyl was used to conjugate the different types of polymer that wrap around
the nanotubes. So, breaking down one by one, might work.
I know that phenylalanine is the 23 added amino acid by Schutz and his gang. It is un natural, so this could be the un natural oligomer. or polymer, at least one of them. It conjugates to the
other new two which are pyrolysine? and selenium We usually have 20 amino acids. These 3 were added. 21, 22 and 23.
thiophenyl:
can see compounds made from this: toulene I believe is in the chemtrails, or was a time back.
so if this replaces toulene? mmmmm
======================
http://www.rdchemicals.com/chemicals.php?mode=details&mol_id=1781
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kritters
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« Reply #19 on May 18, 2012, 10:31pm » | |
May 16, 2012, 11:35pm, skyship wrote:
Lunker,
good test. That would show us something, or tell us how the plaque keeps forming?
make it simple. for sure. Might be all we need.
will give it a try.
Thanks for reminding us again of some of the most simple products. Doesn't have to be as complicated as the Spirochete or fusobacterium link, but wonder what it will do to the polymer
oligomer. Might just eat that too? Does vinegar eat resin?
Since citrus remove the glue spray can remove glue off bottles where labels have been. Could even the shavings of oranges, grapefruit or even lemon assist in breaking down the resin oligomer?
mmm another test to try. Next time I have a filament will put it to the test, see how it reacts. Of course if they are made of some kind of vinyl carbon mix, I don't know. The polymer could be broken down
I bet if it is made of resin?
Skyship |
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Why is everything so timely here with what is going on with my experiences? Speaking of citrus!!! I was cleaning out my closets last week and sorting out my clothes. I came across a few white pieces of clothing I wanted to keep, only they had some rust stains, from who knows where. So obviously I googled to find out what gets rid of rust stains and lo and behold, it's citrus. Some websites right out say to use lemon to rub on the rust and other websites touting commercial rust removers from clothing I also googled and found that citrus is the main ingredient.
So, being at least a few brain cells saving me from being a dummy, I concluded that citric acid is key in HEAVY METAL removal. So, actually, acid is what removes rust.
so, I'm thinking that acid, esp citric acid or the acid in vinegar could be a chelator. If that's true it's awesome but the next step is to find out if it releases the metals into the bloodstream again, or just somehow binds to something and is eliminated.
Kritts
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lilsissy
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« Reply #20 on May 18, 2012, 10:41pm » | |
Very short on time but I have also thought hthis quite possible
fusarium
but also think nano mag type added to refold.
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kritters
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« Reply #21 on May 18, 2012, 10:42pm » | |
Also, the h2o and vinegar thing.
This reminds me of something that could make sense here.
Years ago I read the book, "Vermont Folk Medicine" by Dr. Jarvis MD. The big remedy recommended was the taking of pure raw apple cider vinegar and honey.
I think this is a great remedy for many things, because the honey sugar lures out the pathogens, then the acid in the vinegar terminates them.
THE TERMINATOR!!!!
I've seen this combination of bait and trap suggested as remedies before and it got my attention. Draw them out into the bloodstream or gut or wherever and then attack.
Works for me!
And actually, I think that might be why the wine spit works. Maybe.
Kritts
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kritters
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« Reply #22 on May 20, 2012, 10:30pm » | |
May 18, 2012, 10:41pm, lilsissy wrote:
Very short on time but I have also thought hthis quite possible
fusarium
but also think nano mag type added to refold. |
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LIL SIS!!! So sorry I missed your post! Notice, it was a nano second from mine 
can you expound on your "nano mag type added to refold"?
Kritts
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kritters
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« Reply #23 on May 21, 2012, 7:27pm » | |
ok, here's what's the weird deal of the day.
I come across this article about plant pathology and just for a kick did a search within the article for "fusarium". I wasn't even sure about what we had been discussing, but winged it as I usually do. 10 word associations came up.
I'm not up for reading all of it, but maybe someone else might be.
http://spec.lib.vt.edu/arc/ppws/wingard3.htm
Kritts
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kritters
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« Reply #24 on May 21, 2012, 8:23pm » | |
and how weird is this? I just googled, "fusarium and Lyme disease" and came up with posts I made that I had completely forgotten about.
Kat and Kam, if you're out there and still reading, can you check in? It seems your post was deleted or something on that forum.
http://www.morgellons-disease-research.c....-gm-cotton.html
xoxoKritts
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kritters
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« Reply #25 on May 21, 2012, 8:32pm » | |
Well, this seals it for me. After showing maps of where Lyme, Morgellons, and bedbugs are, and the water elevations, I believe that it is being spread AT LEAST by the water, and MOST DEFINITELY from CHEMTRAILS which leach into the water system which spreads it through sewers, plumbing, whatever.
It's the freaking FUSARIUM in the cotton fields, our water systems, our clothing! I got the exact same fiber photos from my sheets that I got from my skin.
It's just that freaking simple! Aspergillus, e-coli, fusarium, a Burgdorferi spirochete species, and throw in a protein and some bacteria and voila!!!
U gotcher Morgellons.
f**k YOU MONSANTO, YOU f**kING EVIL DEVIL!!!!
KRITTERS
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kritters
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skyship
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« Reply #27 on May 21, 2012, 11:11pm » | |
fusobacterium nucleatum and fusarium are not the same thing.
But, maybe we can search on this; This was started by a dentist and others.
www.homd.org
video, he explains cultivated and uncultivated Oral microbiome and and metagenome and who clones are everywhere, but not properly identified because they have not been named correctly.
We should be able to use this data base to help identify, if not the clones, the un cultivated organisms.
http://www.scivee.tv/node/10023
I will add to reference list.
skyship
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skyship
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« Reply #28 on May 22, 2012, 12:42am » | |
Will see what they have for fusobacterium, can form oligomers(tau) in ALZ, and is from plaque forming from fusobacterium nucleatum on the teeth. It makes an oligomeric motif. or pattern.
fusobacterium nucleatum has nucleaus and does make long filaments, antenna like. This would be the filament formers.
inorganic diphosphatase??????????? well mmmm
http://www.homd.org/modules.php?op=modlo....h ow=prior_link
On top at right can find the fusobacterium this could be the clone then?
fnucp_c_1_3 fusobacterium nucleic protein
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kritters
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« Reply #29 on May 22, 2012, 9:17pm » | |
whatever you say, girlfriend.
I hang on your every word.
What's interesting about this fusarium species is that I see it implicated in teeth (gingivitis) and eyes, and most likely ears. All connected by the tri-geminal nerve and the sinuses.
xoKritts
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