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caroltrew
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 Mitochondrial Antibodies
« Thread Started on Jul 22, 2012, 7:41am »
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Can anyone tell me if we have any post on Mitochondrial. I have tested posted for that, along with Lymes, Microplasma P., Candida Albicans AB and everything that goes along with it, I guess I am having a hard time reading blood results and the Dr. did not bother to sit down and explain everything or anything, should I be surprised? I new I had Morgs. and like every ones been saying 70% or more of Morg. patients have been tested positive for Lymes. This is my second Lymes test. I was tested about 2 yrs. ago and the Dr. (Different Dr.) said nothing showed up, but I don't know what test she used I can't seem to get my records I have asked like 15 times and my GP also. strange, like she was trying to hide something. We did the Western Blot this time and everything came up, I am so relieved now at least we know for sure and can start treatment (of some kind), but I don't know anything about this Mitochondrial Rheumatoidal thing. Does anyone know where I can find some info?
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Carol Trew: Morgellons sufferer searching and learning to treat myself, I have no other choice, I won't give up on trying to rid myself of this horribal new disease. As most all others I have recieved no help from countless Drs. and have been left to treat myself, only with the help and the knowledge of the others on these blessed sites. Thank you all for any help and all information obtained.
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 Re: Mitochondrial Antibodies
« Reply #1 on Jul 22, 2012, 2:00pm »
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Carol,

Yes, Lil Sis and I did some work on that a while back, but, I do think the Rheumatoid Arthritis is involved with the spirochetes. One of our researchers is looking at NAC, some have used this, for the spirochetes in mouth. So, The mitochondria has its own set of genes. that we do know. I think we should delve into this because I think th RA, the ALS, ALZ, the Lymes and even the AIDS are all involved with these newer
spirochetes.

Did you get results from Borrelia and Babesia? Did they actually name the spirochetes? Just in the blood?

Putting it in the form: Mitochondrial Rheumatoidal makes sense.

So, spirochetes of some sort, Microplasma P(Pneumonia?), and Krtts has found the Candida Albicans very much part of this, seems to be part of the biofilm and what forms and works with spirochete. It needs a mucoid base. That and some sort of fungi present, works with the candida. Was a fungi present?

So, if we concentrate on the Mitochondrial Rheumatoid we may find what causes the calcium forms.
Something calcifies.

Here is some info on what some of the abbreviations on the test may mean.

http://labtestsonline.org/understanding/analytes/ama/tab/test

IgG and IgM are important. If we do not have the antibodies or they break down then the disease
or spirochetes are increased sometimes in different form.

This spirochete is the hidden one in Aids as well.

So, somehow way back the mitochondria has changed due to what they call non coding genes or genes from other organisms, viruses, bacteria, or yeasts etc.

but the mito and rheum is a new one on tests as far as I know. They always called any of that OA
or osteoarthritis, when RA is totally different, involves the synovial fluids and smooth muscle.

This may help us get there. I know Dr. Fry is studying RA, ALS ALZ and amyloids. I think this has a lot to do with it.

I will see what I can find. The mitochondria and the synovial fluid. Relates to collagen.

Thank you for sharing that with us.

Skyship



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 Re: Mitochondrial Antibodies
« Reply #2 on Jul 22, 2012, 2:11pm »
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Carol,

Was there any mention of the RF or rheumatoid factor?

" Limitations: 10% to 15% of people with RA will have a negative test.
Rheumatoid Factor (RF)

Rheumatoid factor (RF) is an abnormal antibody that attaches to an immune system protein in the body called immunoglobulin G (IgG), forming a molecule known as an immune complex. The immune complex can trigger different types of inflammation-related processes in the body. This blood test is eventually positive in 70% to 90% of people with rheumatoid arthritis. The results can help confirm a diagnosis if a patient's signs and symptoms are consistent with those of RA. High levels of rheumatoid factor are associated with more severe disease.

Limitations: Because 70% to 90% of people with RA have high levels of rheumatoid factor, that means 10% to 30% do not. Furthermore, RF can be positive with other diseases, including:

chronic hepatitis
chronic viral infection
dermatomyositis
infectious mononucleosis
leukemia
scleroderma
systemic lupus erythematosus (lupus)

In rare instances the blood test is positive in healthy people
http://www.webmd.com/rheumatoid-arthritis/arthritis-test-results

===========



Some of us of lupus like symtoms, and the dermatophytes. in skin and sclerodermis.

So, this RA may be key to finding the core of all of this. I have thought this for a while.

They used to call it "pleurisy, or bursitis". in the past.

It is like having pneumonia in the joints, which would be near the synovials.

If microplasma pneumonia is part of that?

This is interesting:

"The Puzzle of RA Flare-Ups

One of the enduring puzzles of so-called autoimmune disorders is that they all go through cycles of exacerbation and remission. Hundreds of experiments on animals have proved that mycoplasmas are important cofactors in arthritis and other chronic rheumatic disorders and that the tetracycline family of antibiotics suppresses mycoplasmal infections. Immune system control mechanisms appear to be cyclic in nature due to time lags in other regulatory processes (digestion, sleep cycle, etc.)

Mycoplasmas are capable of long-term intercellular in vivo survival and slow, intracellular replication, so they may be resident and waiting for some trauma or barometric pressure changes to activate them when the host’s immune system has moderated its operation. Thus the progression of the infection is cyclical, with waves of reemergence followed by withdrawal to less detectable forms.

Therefore, when mycoplasmas act as antigenic substances, triggering internal allergic responses, they release toxins intermittently to a sensitized area, subsiding and then reappearing. Antibodies move through the body via white blood cells and platelets, and it is through this means that RA migrates from shoulder to hand to knee as the antibodies launch counterattacks against local antigens and toxins. The antibodies move on to new battlefields whenever migrating pathogens like mycoplasmas flare up.

This type of ebb and flow explains the types of flare-ups that RA sufferers describe.



RA Triggers

The apparent causal relationship between changes in the weather and RA pain can be explained. Clinical evidence shows that two environmental factors can cause flare-ups: (1) a sudden drop in barometric pressure and (2) the presence of high humidity in conjunction with this drop. The aches and pains correspond to a sudden release of antigens to a sensitized area, confirming Dr. Brown’s theory that migrating or shape-changing mycoplasmas act as antigenic triggers. As the microorganisms migrate out of the body they stimulate respiratory distress, thus broadcasting their seeds via effluvia (e.g., sneezing) to other mammalian hosts.

Trauma to joints and tendons may cause structural changes, further reducing oxygen transport and limiting removal of fluids and wastes in the region of the injury. The lack of oxygen in the traumatized area increases pressure and swelling. The usual treatment is anti-inflammatory drugs. For the COX-2 enzyme to work, the body needs sufficient copper and zinc. Hyaluronidase enzymes are destructive and facilitate bacterial reproduction. Vitamin C thwarts hyaluronidase

Mycoplasma are borderline anaerobes. That is, they are sensitive to changes in barometric pressure. They move about the body in search of more comfortable places to reside. This movement triggers the inflammation response from the immune system. Hyperbaric oxygen treatments force oxygen into compromised cells and tissue, thus allowing the infection-fighting function of white blood cells to proceed.

Aerobic exercise, deep breathing, use of a slant board are recommended. DMSO and other topical salves help improve circulation to the affected areas. Coenzyme Q10 and anti-oxidant vitamins are worthwhile supplements.

http://www.ra-infection-connection.com/free_articles/MycoRole.htm
==============================


So, if the microplasm is an aneurobe, then we need to give it oxygen, there are oxygen tabs you can get. That with Vit C may help. They live without oxygen, so the more we give it, the less they will thrive. Some use the tetracycline, but, you will have more issues with that and the herx are awful.

This can be done naturally, I believe. What did they suggest for treatment?







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 Re: Mitochondrial Antibodies
« Reply #3 on Jul 22, 2012, 2:16pm »
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LD as in Bb is not the cause of Morgellons, in my opinion. The spirochete is, and it takes on different forms. and affects in different ways. So, which form LD or the new form? I think the new form is related to Morgellons, because they are different sizes and types. In the mouth is treponema, and a fusobacterium of some sort, mixed with gingivitis.. This is not the real LD, which was from Plum Island, but, most likely was modified after that or was recombined.

The main core is the spirochete or worm like form. This worm like form needs mucous. And one of the landing places would be the synocial fluid. Goes where fluid is or glue like fluid.

What we are finding is the core of the amyloids. Something is collecting calcium from bones to survive or the marrow in the bones.

So Mitochondrial RA brings in another type of thing.



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 Re: Mitochondrial Antibodies
« Reply #4 on Jul 22, 2012, 2:31pm »
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Mitochondrial RA.

Collagen and lactic dehydorgenase.

"What Is Cyclooxygenase (COX)?

Cyclooxygenase (COX) is an enzyme that is responsible for the formation of prostanoids. The three main groups of prostanoids -- prostaglandins, prostacyclins, and thromboxanes -- are each involved in the inflammatory response.
Two Forms of Cyclooxygenase (COX)

In the 1990s, researchers discovered that two different COX enzymes existed, now known as COX-1 and COX-2. Cyclooxygenase-1 (COX-1) is known to be present in most tissues. In the gastrointestinal tract, COX-1 maintains the normal lining of the stomach. The enzyme is also involved in kidney and platelet function. Cyclooxygenase-2 (COX-2) is primarily present at sites of inflammation."......

http://osteoarthritis.about.com/od/osteo....looxygenase.htm
=======================================

This has something to do with platelet functions. This was mentioned to me once when I had herx reaction to a flareup of the skin. It covered my face, arms and legs. Was so itchy and painful at the same time. But, once past the herxs it began to heal. Peeled skin but it did heal.



Cyclooxygenase-2 (COX-2). seems to be the enzyme causing the RA.


Morphologic changes in synoviocytes

See Fig. 2. It is in the fibroblasts........ those cells....

http://www.biomedcentral.com/content/pdf/ar1199.pdf

Wow, I do think the findings on RA will tell the whole story. Thank you again for sharing with us.

This mitochondrial enzyme?


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 Re: Mitochondrial Antibodies
« Reply #5 on Jul 22, 2012, 2:43pm »
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Carol,

Got me going on this. I remember talking about this before, something we have from ancient lines.

The mitochodrial is related to our given genes.


so, the core could be this Bcl....

In RA, the synovial lining thickens in part through increased proliferation and/or decreased synovial fibroblast cell death. Here we demonstrate that the anti-apoptotic protein, Bcl-2, is highly expressed in RA compared with osteoarthritis synovial tissues, particularly in the CD68-negative, fibroblast-like synoviocyte population.

http://www.jimmunol.org/content/164/10/5227.full

It gets complicated but if we center on the mito RA and synoviocyte increase, which is the thickening of the walls of the cells. I am having similar issues in gut, so wonder if these actually form the gall stones and kidney stones? Bet they do.

What is Bcl-2? Involves the cytoskeletal cells.
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 Re: Mitochondrial Antibodies
« Reply #6 on Jul 22, 2012, 3:07pm »
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Look up sub mitochondrial particles....seems they have been changing us.

Ultrasound opens the mitochondrial up to manipulation from inorganic phosphorus,
I also believe that the new organelle found in many Morg sufferers, Agrobacterium, may have a role in causing allergy and immune reactions.

Lyme positive may be agrobacterium positive , I have something inside of my cells that is super aggressive to all allergens but I carry high viral loads of Epstein Barr....Herpes type 1.

The Bb ..
Ralph Nader did an article on
Barbara Johnson from the C.D.C, owning the genetic sequence to it, there-by stopping research into Bb.

Also on the Russel Tice Wiki Page ,
there was once references to Animal Bb being mixed with Lyme Disease and released unto the public under the Ford Administration. He also said that Americans should not know for over 200 years what .N.S.A. what Gov programs have done .
I think Signet is big in it...N.S.A..... DARPA....SATE LIGHTS they are controlled by Homeland Security who looks to A.I. for most of it's intelligence gathering . Homeland Security dictates to all including the C.D.C. ,N.I.H. and Congress obviously per the Russel Tice story, also.
The C.D.C. is also a participant in the World Health Organization, G.L.E.W.S. agreement. The agreement among member nations has limits and rules about announcing to the public the emergence of new pathogens


Part of the quotes on Russel Tice Wiki Pages where removed after we ran threads calling attention to it but Rumor mill News has old threads still with original Quotes that came into question.


David Larson versus Alfred Mann , The suit mentions Russell Tice
( whistle blower) from the N.S.A. on page 6. It seems Dave and Russ have worked together in the past.

David Larson helped in the development of intracellular devices that can be delivered by needle injection, these are then Daisey's chained together by what seems to be molecular wiring ( Morg Fibers ).
These Fibers A.K.A. Molecular wires are most likely to be found in one of the many existing patents available on line at the U.S.P.T.O. .

WE are searching for the cause of a new beast,
so I don't think Biologically traditional ones will apply in singular form in Morgellons Sufferers but that we will continue to find that they used a new mix of old polymers or what is termed.....
Co-polymers ( natural and Synthetic ), now grow in many of us symbiotically.
While other natural D.N.A. halo types ( Morgellons Sufferers) are inflicted by these termed .....

"Daisey chains " ET AL... David Larson versus Alfred Mann.

These molecular chains do not mix well with certain halo types and or existing pathogens acquired since birth there-by causing illness and disease.

These co-polymers, are reconfigured and refolded for the conquest over our biological make-up (to rule by wireless energy) the will of man.


There is an unseen race
(RACE, as in to win but maybe both race of beings)

to rule over an artificially created nervous system. One that reaches from every brain neuron to every muscle cell in the body via the vascular bed, crossing also the blood brain barrier . The brain can be controlled by computer software systems.
Our brain is a biological computer can be programmed via wireless energy.

Napoleon and Hitler did not advance in the world of the small but ruled with war, very visible. The whole world was aware was of their evil.
Now it is possible with this new technology-biology-synthetic life- mix to rule mankind by changing mankind's molecular make-up and tapping into NEVER BEFORE SEEN STATE of sub- consciousness -
a computer controllable artificial state..... as stated in the Human Brain Machine patent.

This conquest continues in the unseeable world of the small without alerting the masses, even the minority of mankind are blind to the effects they feel in their bodies.
Morgies and certain other watchers of science and madmen continue to investigate and see through computer imaging the resident evil that has been delivered into our bodies through chemistry.

The gate of man's soul via our molecular make-up is being manipulated.
Corporations that change and rearrange D.N.A. such as Monsanto are reaching for these apples....us.

For the record,
many of my test results are unobtainable, I have repeated blood tests some 3 times and I hear excuses like they didn't take enough blood or the lab messed up , over and over again.

Yes, I feel certain that a network of Hospitals and Medical Academia institutions are practicing only acceptable science. This exist much like they Illuminati and Morgellons...... not to be seen easily.

The people who run the dog and pony,
Medically Accepted Practices have control over Corporations.....
B.C.B.S. , Medicare, Medicaid are influenced and have influence , money...
The money changers have entered not only the Temple of God but also the temple of man..... by chemistry.
These people buy and sell agendas at the finest restaurants with their egos and expense accounts.

Reminds me of the days when getting a job through the Carpenters Union involved being in with the click, going to the bar....commoradary.

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 Re: Mitochondrial Antibodies
« Reply #7 on Jul 22, 2012, 3:17pm »
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Bcl-2 same as COX-2? They sure make the deciphering of this very difficult, calling it all kinds of things.



Apoptosis is cell death of inflamed cells, supposedly, but I believe it kills regular cells as well.
Here is core. Again, every test we have that indicates some of this is so important, Carol.
So glad we can look at this again, the cause of metasis in breasts, as well seems to be same, is estrogen related, hormones. As many researchers are finding. So, the membrane of cell is the protein that lets this into those inflamed cells and the regular non inflamed cells.

===================================

During Apoptosis Bcl-2 Changes Membrane Topology at Both the Endoplasmic Reticulum and Mitochondria


http://www.jimmunol.org/content/164/10/5227.full

================

See where the Bcl-2 is: It destroys our natural mitochondria: Our natural EVE.


http://origin-ars.els-cdn.com/content/image/1-s2.0-S0278691512003705-fx1.jpg


Lil sis, seems Carol may have key to submicron?

Bless her heart!


Skyship
Yes, Lil Sis, the submicron particle, same thing Carnicom is finding.
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 Re: Mitochondrial Antibodies
« Reply #8 on Jul 22, 2012, 3:28pm »
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Does it start with this?


http://www.microscope-microscope.org/app....eba-proteus.htm


the protozoan? under Dr. Fry's biofilm?


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 Re: Mitochondrial Antibodies
« Reply #9 on Jul 22, 2012, 3:31pm »
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Lil Sis,


David Larson helped in the development of intracellular devise that can be delivered by needle injection which are then Daisey's chained together by what seems to be molecular wiring ( Morg Fibers )


That is where the "Daisy" comes in. Mentioned many times in some people's treatment. Treat them for the 'Daisy Syndrome" or Morgellons.


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 Re: Mitochondrial Antibodies
« Reply #10 on Jul 22, 2012, 3:34pm »
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Think of our DNA as now hybrizing those homologous proteins from other organisms. This may have happened in past, but, why is it being done now? Some of us will go back to the earlier form of this "evolved form". called "de novo". We will go back to the "rock". Is that not what is happening to us?

We are hardening stone inside of us. The particulate matter, the submicron particle of organic form, then the "submicron particle" of nanoparticle form, bottom up. So to tackle this we have to see what "building bricks" were used.

and on molecular level. the molecular forms and the wiring.




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 Re: Mitochondrial Antibodies
« Reply #11 on Jul 22, 2012, 3:37pm »
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This deletes our original DNA. that is how it is done. And, this only preserves certain select of certain select psychotic humans. So agreed, Lil sis.

We fight for the "natural" even if it means we find out how to adapt backwards, to the natural form again we were given. Not the new forced/adaptive that fits on the elite.


God Bless us each and everyone, for the power knows!

The spiritual wars through physcial manifestation is now here.

How do we gain the power to adapt backwards, to our God? For he was the first "Rock".



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 Re: Mitochondrial Antibodies
« Reply #12 on Jul 22, 2012, 3:39pm »
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What was sprayed on Palestine? Phosphorus. What was in Sodom and Gomorrah?

What is in the genes of those perpetuating this horror?

What do they want?

They do not have what we have, so they have set about destroying our very mitochondria, our original Eve mitochondria for the sake of the alias Eve.

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 Re: Mitochondrial Antibodies
« Reply #13 on Jul 22, 2012, 3:42pm »
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They cannot adapt to heat, or heat shock proteins. So, they alter those who can. Meanwhile, protecting themselves. It is in the barometric pressure, the heat shock factor. Many of us see this. We know the global warming was conceived to make this happen. So heat the earth. Takes down a few ethnic groups.

the real Sons and daughters of God, of Adam and first Eve, not the second Eve. Therein the mito eve factor was first to go. RA, ALS, ALZ etc.......



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« Reply #14 on Jul 22, 2012, 3:49pm »
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So, Lil sis, we need to change the sound? The harmonics, the resonance, the music of our genes.


sub mitochondrial particles.


In the hydrogenase: this was what is called dehydrogenase, taking hyrdrogen out. of our DNA. that is what hold the dna together. There is an artist who has this down will find those 3 panels. It gives us a clue as to what is being done.


Sub-mitochondrial particles

Treatment of mitochondria with ultrasonic vibrations under appropriate conditions tears open the membranes and leads to the formation of tiny inside-out vesicles derived from the inner membrane, containing trapped cytochrome c originating from the inter-membrane space. Sub-mitochondrial particles may retain partial coupling between electron transport and ADP phosphorylation. They have been much used for experimental work because their inverted membrane orientation provides direct access to the respiratory chain without the complications introduced by the substrate transport systems.

Two activities which are most easily studied in sub-mitochondrial particles (although they also occur with intact mitochondria) are the energy linked transhydrogenase and reversed electron transport.

The energy-linked transhydrogenase: This bizarre membrane-spanning enzyme catalyses the reversible transfer of reducing equivalents between the mitochondrial NADPH + NADP pool and the NADH + NAD pool, while simultaneously pumping 2 protons and 2 charges across the inner mitochondrial membrane. The energy linkage keeps the NADPH/NADP couple about 500 times more reduced than the NADH/NAD couple, despite the exact equivalence of their standard redox potentials.

NADPH + NAD+ + 2H+(inside) <=> NADP+ + NADH + 2H+(outside)

It is not clear which direction the transhydrogenase takes in vivo. Enzymes reacting with NADP generally have a more negative redox potential than those reacting with NAD, except for glutamate dehydrogenase, which is the only enzyme with a dual coenzyme specificity. This is a problem, since the dual coenzyme specificity should lead to an energy-wasting futile cycle if the two coenzyme pools are at different redox potentials.

The arrangement must confer some selective advantage (having persisted unchanged for 2000 million years!) but the biological benefits remain obscure. It may involve the overall regulation of nitrogen balance, by using the ammonia-fixing reaction with NADPH to partially counteract the ammonia formation with NAD, but this hypothesis remains highly speculative. Mitchell once suggested that the NADPH-linked pathways for isocitrate, malate and glutamate were equivalent to fifth gear on a car, giving enhanced ATP yields and better fuel economy at the expense of snappy performance. When maximum ATP flux was required the ADP-mediated activation of the NAD-linked pathways would "drop down a gear" to enter the overtaking lane.

Reversed electron transport: Oxidative phosphorylation is a partially reversible process and in the presence of an artificially high ATP/ADP ratio electrons from a weak reducing agent like succinate can be forced backwards through the respiratory chain carriers to yield a stronger reductant such as NADH:

succinate + NAD+ + energy => fumarate + NADH + H+ (overall reaction)

http://www.bmb.leeds.ac.uk/illingworth/oxphos/particle.htm
===============



We can do this. we need the right haromic, resonance, and music of our real DNA to overcome this.
How do we find the music, the chords, the sounds? In the beginning was the WORD. somewhere in there. It was spoken.


look at the actinospherium:
http://www.microscope-microscope.org/app....a/sarcodina.htm

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 Re: Mitochondrial Antibodies
« Reply #15 on Jul 22, 2012, 3:50pm »
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Ultra sound vibrations will knock these suckers out, vit C and oxygen. Hit em at the core, if we can.

I bet ultrasound will show these for what they are.

If Ultrasound can find gall stones and kidneystones and give us the size?

Wahlaaaaaaaaaa!

Tell me they cannot see these?
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 Re: Mitochondrial Antibodies
« Reply #16 on Jul 23, 2012, 12:47pm »
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Check this out. I just received it in Dr. Mercola's newsletter. Don't know if it has anything to do with this topic, but Dr. Shoemaker talks about mold, spirochetes, etc. and something to help against these pathogenic culprits.

Kritts
http://articles.mercola.com/sites/articl....0722_SN L_Art_1
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 Re: Mitochondrial Antibodies
« Reply #17 on Jul 24, 2012, 2:31am »
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He does not have treatment for the dinoflagellates though. They live in the algae, they are not the Algae. " Pfiesteria, a dinoflagellate (like an algae) " Not really, it lives in red algae. Algae is more the problem then mold. We know black mold, and it is a water mold, Heat shock proteins were made from them.

Too costly, and most of us have already cleaned up the mold, so that is not what causes Morgellons.

It may lead to it though, so he does have a point there.

Also there is a Mold Hoax as well according to these folks. Remember, he gets to be shown all over TV.

http://www.deepdyve.com/lp/hindawi-publi....hoax-asw2xQ1zrf

just to be aware, we are on top of this.

http://www.advancedmoldinspections.com/mold_scams.html


I don't like the "chronic concept", that means it never goes away. However, he has some good ideas,
and some help for Lymes. As long as it is herbal, natural and non clostridium forming. hey its good.

If mitochondria is altered by Heat shock proteins, and those are in this mold, spirochete dinoflagellate, the water would be the issue.

A look at watermolds with this diatoms, dinaoflagellate in the algae. might be good.




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 Re: Mitochondrial Antibodies
« Reply #18 on Jul 24, 2012, 7:35am »
[Quote]




Sky said,

IgG and IgM are important.


For me the IGE blood test the Pain Clinic Doctor ran on me really helped me. It helped to cut down on the spiraling flu symptoms my life was revolving around on.

This Dr. had attended a symposium on Fibromyalgia and he said the symposium brought my symptoms to his attention.

When I went for a visit afterward , I asked him to run a test for lead.
Well, he instead did a IGE Food Allergy test.
He had a panel with 8 different Food
Corn
Soy
Wheat
Egg
Milk
Peanut
Shellfish
Fish -not shellfish

0 is no reaction
it goes up to 6
being as high as it gets.
6 is in the anaphalitic shock reaction reaction....

I was a 4 and higher in all groups.

I had not considered food allergy as contributing to the pain I had been experiencing , flu like -all over...muscle...joints...slight fever..loss of energy
He explained to me that my body sent out messages to attack these foods after they had passed through my bowel, which can be up to 48 hours after consumption, so I did not put together the effect they where having on my body.

I try my best to avoid these Foods , some make me ill right away while others sneak up and wrestle me out of it.

IGE for food is an easy test and may show why your body is run down and not able to fight off others invaders.

I seem to have had an allergy to aspirin since birth, mom said it made me run a fever.
I remember sneaking them out of the bottle and eating them, I craved aspirin....
Cravings can also be a sign of allergy.

I am certain I have a high immune system that is harm full to my own cells.
I am certain I have an allergy to Florescent Lighting,
I turn purple after about 30 minutes and start having mental changes such as drowsiness and then flip to feeling very anxious under Florescent lights.

I believe I may be failing to wireless energy.

I never have been diagnosed with Bi-polar but certain lighting can and does flip my body which includes the brain into undesirable states.
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 Re: Mitochondrial Antibodies
« Reply #19 on Jul 24, 2012, 8:10am »
[Quote]



Sky I have been skipping around this post so
Well ....
went into the kitchen to butter a biscuit when I considered
again, why do I turn purple-blue under F.Lighting
again my mind went back to my daughter Jacqueline going on Ancestry on line and hitting the back button on my Great- Grandfather Richard Green Hackworth....all the way back to King Herod. RGH, was a direct father to son , all male descendant of Herod , his daughter was Myrtle Emmaline Hackworth..my Grandmother.

It is said that King Herod visited the dead sea often and was plagued with worms.

I was considering the Stanford University Light and Brain Control experiments. Wonder if I was slipped a gene through a
trans-human engineering during the Ford Administration, as inferred by the former Russel Tice Wiki page or born reacting to it by an insert retro-viral type by old crazy Herod.

If Hitler was studying the heavy water where Herod floated.

The dead sea , it's layers have flipped...why.. forgot..
Why do bird mites that where once exto..why did they go endo..why

Ancient Alchemy , the tree, the apple...the apple computer...gene sequencing..the past seems...the garden seems to have had 2 very
interesting trees.

Also I recall reading of some experiments that where admitted to, of an Air Born Ariel Scattering Experiment ( Bacillus , I believe ?) over Detroit and Canada was it the 50's or the 60's ?

I am convinced I have a conversion of the blood under F. lighting .
I first was convinced of this when I was in middle school , Ms. Rice's class. Ms. Rice would freak out sometimes real bad climbing on top her deak screaming.
That is not a co-incedence , I think she too was having a hard time dealing with the lighting , they seems to have bad ballasts on them.





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 Re: Mitochondrial Antibodies
« Reply #20 on Jul 24, 2012, 8:30am »
[Quote]



This was proved as far as I am concerned, the agro-connection I mean,
this was used Agrobacterium C-58, the one that is 1000 times stronger that the other agrobacteriums.

http://www.ehow.com/about_6313255_agrobacterium-morgellons-disease.html
Agro was used on our food and then found to change genes in humans
Agriculture
Human culture
The Garden
The Trees

I used to climb the Mulberry in my backyard as a young child,
I loved the Lil yellow caterpillars that would rustle and tickle my body.

Remember the silk industry, they changed the silk worm by mixing it with a different species way back in the 1600's.

Trees, worms, mixing cotton with wool?
maybe with silk also?

Agro
The mixing, the root grows hmmm
The tree's
Wasn't the tree of life encased in a ring of fire so that Adam's line, being as it was fallen could not reach eternal life and live forever in the fallen state...estate?

How do we unscramble eggs?

The orginal Code must be reset, I belive they...we... as beings... have changed our garden and broken the Code.

This is inserted............ however, when ever, inserted...snuck in...
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 Re: Mitochondrial Antibodies
« Reply #21 on Jul 24, 2012, 9:53am »
[Quote]



Latest om mtdna

http://www.wellcome.ac.uk/News/2012/Features/WTVM055438.htm

so agro makes sense


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 Re: Mitochondrial Antibodies
« Reply #22 on Jul 24, 2012, 10:22am »
[Quote]



http://udini.proquest.com/view/towards-delivery-of-dna-into-pqid:1865667421/
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 Re: Mitochondrial Antibodies
« Reply #23 on Jul 24, 2012, 10:17pm »
[Quote]


http://udini.proquest.com/view/towards-delivery-of-dna-into-pqid:1865667421/

does this work?

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 Re: Mitochondrial Antibodies
« Reply #24 on Jul 24, 2012, 10:50pm »
[Quote]


Seems one can't get to the book itself. So, must be something there, we are not supposed to decipher. Works the same:

http://www.ncbi.nlm.nih.gov/books/NBK21942/


Lil sis these leads up to the [b]biotinylated PNA
. peptides. pseudopeptides....... into the mito.

The recombination theorys and workable transposons came from Lederberg and Tatum.
[/b]

"Do bacteria possess any processes similar to sexual reproduction and recombination? The question was answered in 1946 by the elegantly simple experimental work of Joshua Lederberg and Edward Tatum, who studied two strains of Escherichia coli with different nutritional requirements. Strain A would grow on a minimal medium only if the medium were supplemented with methionine and biotin; strain B would grow on a minimal medium only if it were supplemented with threonine, leucine, and thiamine. Thus, we can designate strain A as met− bio− thr+ leu+ thi+ and strain B as met+ bio+ thr− leu− thi−. Figure 7-2a displays in simplified form the concept of their experiment. Here, strains A and B are mixed together, and some of the progeny are now wild type, having regained the ability to grow without added nutrients. Figure 7-2b illustrates their experiment in more detail. "............

Demonstration by Lederberg and Tatum of genetic recombination between bacterial cells. Cells of type A or type B cannot grow on an unsupplemented (minimal) medium (MM), because A and B each carry mutations that cause the inability to synthesize constituents (more...)
http://www.ncbi.nlm.nih.gov/books/NBK21942/figure/A1306/?report=objectonly

gets complicated, but if can be done in lab, can be done not only in the environment, but whatever bacteria conjugates there and inside gut, utilizing the e-coli there determines what 3rd form is morphed within the gut, lung or skin.

So, delivery can be by air, by chemtrail, or by growing forms in the environment all around us. We become part of that environment. It becomes part of us. The universal genes. Our own mito is no longer our [i]own. It has been removed, overpowered by the bacterial conjugations.


I believe the GlcNAC is what does this:
This study talks of the lectins in different foods, those have been altered with agro? possibly?[/i]

" Peanut agglutinin (PNA), Soybean agglutinin (SBA), Wheat germ agglutinin (WGA), Lens culinaris (LcL), Laburnum anagyroides (LAL). Presence of N- and O-glycans in the composition of proteins of plasmatic membranes of a thin gut was shown. Sialic acids were absent in the composition of alkaline phosphatase of mature mucous production. The expression of lectin-binding glycoconjugates exhibited differences between certain types of chronic thin gut inflammation. WGA with affinity for GlcNAc and neuraminic acid labelled the cells with early thin gut inflammation but most intensely those of fibroblastic type. Staining with PNA and SBA, which are GalNAc specific, were strongly positive. Enhanced PNA reactivity reflected mainly cytoarchitectural pattern. These results documented the heterogeneous glycosylation pattern in different subtypes of thin gut inflammation and indicate the usefulness of lectins in the early evaluation of pluripotential differentiation of thin gut secretory cells..

http://www.fasebj.org/cgi/content/meeting_abstract/22/1_MeetingAbstracts/898.3
==========================================


One thing, Lil sis regarding the pancreas, kidney and gallbladder, is this accumulation of these lectins, they need secretery (mucous) membranes to work. So, can attack to wall of intestine, and perforations of the intestines often come before the pancreas issue manifests itself.



So, the GlcNAC and biotinylated PNA>

may have to do with photoreceptor cells.


s

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 Re: Mitochondrial Antibodies
« Reply #25 on Jul 24, 2012, 11:01pm »
[Quote]


Trying to stay focused on the bacterial conjugation. If that is there and phages are used to introduce other dna, this would be core for replication. The phage would replicate to go after the bacterias. However, if the two bacterias form a 3rd it will still be there, the phages not specific to the new form, only the separate non conjugated forms before the act.





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 Re: Mitochondrial Antibodies
« Reply #26 on Jul 24, 2012, 11:08pm »
[Quote]

So mitochondrial DNA is not the same as "nucear" dna. So, if we are looking just at mitochondrial in RA
and other related issues in Morgellons, that particular conjugation would be different than nuclear.


Lil sis,
Like Kammy says it is the "circular genome". We are linear, Bacteria is circular.


Figure 2. Representative FERTILITY PLASMID. A fertility plasmid carries the genes for conjugation as well as a number of other genes. In this figure the fertility plasmid also carries antibiotic resistant genes.

Before DNA exchange can be discussed it is necessary to understand what PLASMIDS are. Plasmids are best thought of as

Bacgen40.gif (3477 bytes)
Figure 3. Plasmids in a bacterial host cell. A cell may contain no plasmids, one plasmid or many copies of a plasmid. A single host may contain a number of different plasmids (green, blue & pink).

MINI-CHROMOSOMES. Plasmids are composed of DNA which usually exists as a CIRCULAR MOLECULE, only much SMALLER than the genomic DNA (Fig. 2). Click here for a discussion of plasmid vectors and their component parts.

Plasmids vary in size, but most are between 1,000 to 25,000 base pairs vs. 4,000,000 bp in the genome. Plasmids REPLICATE AUTONOMOUSLY from the genomic chromosome. Often there are MANY PLASMID COPIES present in one cell. Further, a cell may contain SEVERAL DIFFERENT PLASMIDS or it may contain NO PLASMIDS at all. Plasmids generally carry genes that are NOT

ESSENTIAL for a cell's survival except under special circumstances. For example, many plasmids carry genes for ANTIBIOTIC RESISTANCE. When these plasmids are present in a cell, it is unaffected by the appropriate antibiotic, but if the plasmid and its antibiotic resistant gene is lost, the host cell becomes sensitive to a given antibiotic. Some plasmids carry resistance genes to several antibiotics, making them very dangerous pathogens. In other cases plasmids, called VIRULENCE-PLASMIDS, carry #VIRULENCE GENES that enhance a host's ability to cause a disease. That is, a bacterium carrying a plasmid containing the virulence gene is able to CAUSE A DISEASE, but when the plasmid is missing that same bacterium is unable to produce that disease. One such plasmid-based disease of recent concern is the strain of E. coli #O157:H7 that produces a severe food-borne disease. We will learn more about this organism in the section on FOOD MICROBIOLOGY. Other plasmids carry genes for protecting a cell against DELETERIOUS substances like mercury, copper or they may carry genes that make it possible for a cell to metabolize an UNUSUAL SUBSTRATE, such as gasoline, as a nutrient or energy source.

The question naturally arises as to the PURPOSE of these plasmids in the evolutionary scheme. The current explanation is that plasmids constitute an EXTRA POOL OF GENE ALLELES and thus enlarge the effective gene pool of the population. Remember that the genome of prokaryotes carries only enough information for between 1,000 to 5,000 genes. But, as we've already learned, the more variety the better a species' chances of #survival are in a fickle universe. The phenomenon of ANTIBIOTIC

This would be like someone afraid of being robbed carrying around an AK-47, a rocket launcher and a small cannon; they might be safe from thieves, but all that bulk and weight is going to seriously interfere with their everyday lives--like getting dates.

RESISTANCE is a case in point. Antibiotics, being natural products of certain organisms, are never-the-less unlikely to be encountered very often in quantities that endanger susceptible sensitive strains, so there is no need to carry resistance genes against the hundreds of antibiotics that lurk in the nooks 'n crannies of the environment. Indeed, to do so would likely tie up all your genes just for this one purpose; clearly not a survival plus.

http://www.slic2.wsu.edu:82/hurlbert/micro101/pages/Chap9.html

Plasmids have one other very significant role to play in this story. They serve as the VEHICLES for carrying genes between cells in the #genetic engineering revolution.


the plasmids,

"

Legend:

Plasmids are similar to viruses, but lack a protein coat and cannot move from cell to cell in the same fashion as a virus.

Plasmid vectors are small circular molecules of double stranded DNA derived from natural plasmids that occur in bacterial cells. A piece of DNA can be inserted into a plasmid if both the circular plasmid and the source of DNA have recognition sites for the same restriction endonuclease.

The plasmid and the foreign DNA are cut by this restriction endonuclease (EcoRI in this example) producing intermediates with sticky and complementary ends. Those two intermediates recombine by base-pairing and are linked by the action of DNA ligase. A new plasmid containing the foreign DNA as an insert is obtained. A few mismatches occur, producing an undesirable recombinant."..............

http://www.accessexcellence.org/RC/VL/GG/inserting.php

========================
So these intermediates are what could form from the conjugates. This can happen with fungi mycelia as well, algae genes, viral genes etc.
But, the phage is important as a vector, If not the bug, then the phage then the nanoparticle. Just upping the anty.
====================

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 Re: Mitochondrial Antibodies
« Reply #27 on Jul 24, 2012, 11:27pm »
[Quote]

Carol,

One question keeps floating in my mind. Why mitochondrial antibodies present? that means these antibodies are removing the mitochondria, our given dna. In epigenetics this is not suppose to happen, this means epigenetics is the selection process. Removing original mito by using anti mitochondrial mechanisms.


What does that? I know this sounds like gobbly gook, but there is something here that is being found.


"Pemphigus vulgaris (PV) is an autoimmune disease affecting skin and mucosa brought on by a loss of epidermal cohesion due to detachment of keratinocytes. In a process termed “apoptolysis,” autoantibodies react with desmosomal proteins like desmoglein 3 (Dsg3) to activate various signaling kinases within keratinocytes, causing their shrinkage, detachment from neighboring cells, and eventual death. In addition to external apoptotic pathways, the activation of caspases during PV indicates a role for intrinsic apoptosis, although how this pathway gets activated is less clear. In this Paper of the Week, Steve Marchenko and colleagues find that a subset of PV autoantibodies can enter the cells and directly target mitochondrial antigens, triggering cytochrome c release and activation of c-Jun N-terminal kinase (JNK) and late p38 mitogen-activated protein kinase (p38 MAPK) signaling."

http://www.jbc.org/content/285/6/e99914.full

============


It is called apoptolysis: can cause other cells to die.

"Together, these findings highlight that apoptolysis is a complex process initiated by at least three classes of autoantibodies targeting desmosomal, mitochondrial, and other keratinocyte autoantigens to trigger two different apoptosis pathways. "...............

Notice the M above the cell damage?

http://www.jbc.org/content/285/6/e99914.full

===========================

remember the study by Stricker and Middleveen? relation to the cattle hoof disease?




this means latent dna, so called non coding dna, is made active for better or worse. some can help, but by activating non coding that only activate when needed, this is over doing it.
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 Re: Mitochondrial Antibodies
« Reply #28 on Jul 24, 2012, 11:36pm »
[Quote]


"Pemphigus vulgaris

Morgellons may be a form of this:

http://www.theestheticclinic.com/pemphigus_vulgaris.html

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